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TRPV4 Channel Agonist GSK1016790A Induces Urinary Bladder Contraction and Hyperactivity

Posted on May 23, 2008 by crimsoncanary

GSK’s transient receptor potential vanilloid 4 (TRPV4) agonist GSK1016790A contracts mouse bladders in vitro and stimulated bladder overactivity in vivo

Filed under: Ion Channel | Tagged: 1016790, agonist, Glaxosmithkline, GSK, GSK-1016790A, GSK1016790A, TRPV4

« Newron completes Phase III recruitment for Safinamide in Parkinsons US and WO patent update for the week ending 05/23 »

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